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Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects.

PaperCitations, authors & open-access status

Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. is a paper indexed in PICO Portal from OpenAlex.

How many citations does Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. have?

PICO Portalcitation count

Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. has 314 citations in the OpenAlex counts PICO Portal stores.

When was Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. published?

PICO Portalyear

Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. has 2007 year in the PICO Portal index.

What is the open-access status of Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects.?

PICO Portaloa status

Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. has green oa status in the PICO Portal index.

What is the DOI of Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects.?

PICO Portaldoi

The DOI for Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. is on the source record.

What is the PMID of Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects.?

PICO Portalpmid

The PMID for Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. is on the source record.

What is the OpenAlex record for Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects.?

PICO Portalopenalex

The OpenAlex for Sulforaphane retards the growth of human PC-3 xenografts and inhibits HDAC activity in human subjects. is on the source record.

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