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Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation

PaperCitations, authors & open-access status

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is a paper indexed in PICO Portal from OpenAlex.

How many citations does Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation have?

PICO Portalcitation count

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has 28 citations in the OpenAlex counts PICO Portal stores.

When was Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation published?

PICO Portalyear

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has 2023 year in the PICO Portal index.

What is the open-access status of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

PICO Portaloa status

Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation has hybrid oa status in the PICO Portal index.

What is the DOI of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

PICO Portaldoi

The DOI for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

What is the PMID of Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

PICO Portalpmid

The PMID for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

What is the OpenAlex record for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation?

PICO Portalopenalex

The OpenAlex for Reduced binding of apoE4 to complement factor H promotes amyloid‐β oligomerization and neuroinflammation is on the source record.

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