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Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist

PaperCitations, authors & open-access status

Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist is a paper indexed in PICO Portal from OpenAlex.

How many citations does Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist have?

PICO Portalcitation count

Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist has 132 citations in the OpenAlex counts PICO Portal stores.

When was Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist published?

PICO Portalyear

Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist has 1989 year in the PICO Portal index.

What is the open-access status of Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist?

PICO Portaloa status

Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist has closed oa status in the PICO Portal index.

What is the DOI of Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist?

PICO Portaldoi

The DOI for Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist is on the source record.

What is the PMID of Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist?

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The PMID for Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist is on the source record.

What is the OpenAlex record for Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist?

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The OpenAlex for Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist is on the source record.

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