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Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques

PaperCitations, authors & open-access status

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is a paper indexed in PICO Portal from OpenAlex.

How many citations does Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques have?

PICO Portalcitation count

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has 33 citations in the OpenAlex counts PICO Portal stores.

When was Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques published?

PICO Portalyear

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has 2015 year in the PICO Portal index.

What is the open-access status of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

PICO Portaloa status

Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques has bronze oa status in the PICO Portal index.

What is the DOI of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

PICO Portaldoi

The DOI for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

What is the PMID of Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

PICO Portalpmid

The PMID for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

What is the OpenAlex record for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques?

PICO Portalopenalex

The OpenAlex for Nanotherapeutics for inhibition of atherogenesis and modulation of inflammation in atherosclerotic plaques is on the source record.

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