M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma is a paper indexed in PICO Portal from OpenAlex.
How many citations does M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma have?
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma has 146 citations in the OpenAlex counts PICO Portal stores.
When was M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma published?
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma has 2023 year in the PICO Portal index.
What is the open-access status of M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma?
M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma has gold oa status in the PICO Portal index.
What is the DOI of M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma?
The DOI for M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma is on the source record.
What is the PMID of M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma?
The PMID for M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma is on the source record.
What is the OpenAlex record for M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma?
The OpenAlex for M2 macrophage-secreted exosomes promote metastasis and increase vascular permeability in hepatocellular carcinoma is on the source record.