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CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response

PaperCitations, authors & open-access status

CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response is a paper indexed in PICO Portal from OpenAlex.

How many citations does CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response have?

PICO Portalcitation count

CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response has 14 citations in the OpenAlex counts PICO Portal stores.

When was CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response published?

PICO Portalyear

CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response has 2020 year in the PICO Portal index.

What is the open-access status of CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response?

PICO Portaloa status

CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response has green oa status in the PICO Portal index.

What is the DOI of CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response?

PICO Portaldoi

The DOI for CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response is on the source record.

What is the PMID of CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response?

PICO Portalpmid

The PMID for CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response is on the source record.

What is the OpenAlex record for CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response?

PICO Portalopenalex

The OpenAlex for CdSe/ZnS quantum dots exhibited nephrotoxicity through mediating oxidative damage and inflammatory response is on the source record.

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