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Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism

PaperCitations, authors & open-access status

Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism is a paper indexed in PICO Portal from OpenAlex.

How many citations does Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism have?

PICO Portalcitation count

Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism has 66 citations in the OpenAlex counts PICO Portal stores.

When was Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism published?

PICO Portalyear

Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism has 2018 year in the PICO Portal index.

What is the open-access status of Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism?

PICO Portaloa status

Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism has bronze oa status in the PICO Portal index.

What is the DOI of Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism?

PICO Portaldoi

The DOI for Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism is on the source record.

What is the PMID of Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism?

PICO Portalpmid

The PMID for Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism is on the source record.

What is the OpenAlex record for Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism?

PICO Portalopenalex

The OpenAlex for Aspirin inhibits the proliferation of hepatoma cells through controlling GLUT1-mediated glucose metabolism is on the source record.

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