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Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation

PaperCitations, authors & open-access status

Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation is a paper indexed in PICO Portal from OpenAlex.

How many citations does Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation have?

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Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation has 919 citations in the OpenAlex counts PICO Portal stores.

When was Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation published?

PICO Portalyear

Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation has 2006 year in the PICO Portal index.

What is the open-access status of Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation?

PICO Portaloa status

Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation has closed oa status in the PICO Portal index.

What is the DOI of Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation?

PICO Portaldoi

The DOI for Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation is on the source record.

What is the PMID of Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation?

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The PMID for Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation is on the source record.

What is the OpenAlex record for Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation?

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The OpenAlex for Angiopoietin-2 sensitizes endothelial cells to TNF-α and has a crucial role in the induction of inflammation is on the source record.

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